Geometry.Net - Health_Conditions: Cerebral Edema

Cerebral Edema
cerebral edema. cerebral edema. Most Common Cause cerebral edema is themost feared complication of DKA in children. It frequently occurs
http://gut1.peds.uiowa.edu/DKA/tsld017.htm
Cerebral Edema
cerebral edema is the most feared complication of DKA in children. It frequentlyoccurs when things are looking better and the medical team begins to relax.
http://gut1.peds.uiowa.edu/DKA/sld017.htm
(Acute) Cerebral Edema
(Acute) cerebral edema This is a cross section of brain which is suffuring fromunilateral cerebral edema. Which side is edematous? How can you tell?
http://www.vet.ohio-state.edu/docs/vetbio550.01/vascular/circulation/cerebral.ht
Edema Cerebral. Edema Cerebral
Brain Edema and cerebral edema, November 11, 2002 New Treatments for Brain Edemaand cerebral edema, November 11, 2002 HONselect ENFERMEDADES CEREBRALES
http://www.cerebralpalsy.de/edema-cerebral.asp
NEJM -- Risk Factors For Cerebral Edema In Children With Diabetic Ketoacidosis
Original Article from The New England Journal of Medicine Risk Factorsfor cerebral edema in Children with Diabetic Ketoacidosis.
http://content.nejm.org/cgi/content/short/344/4/264

Extractions: Nicole Glaser, M.D., Peter Barnett, M.B., B.S., Ian McCaslin, M.D., David Nelson, M.D., Jennifer Trainor, M.D., Jeffrey Louie, M.D., Francine Kaufman, M.D., Kimberly Quayle, M.D., Mark Roback, M.D., Richard Malley, M.D., Nathan Kuppermann, M.D., M.P.H., for The Pediatric Emergency Medicine Collaborative Research Committee of the American Academy of Pediatrics Table of Contents Full Text of this article PDF of this article Editors' Summaries Related editorials in the Journal: Dunger, D. B. Find Similar Articles in the Journal Notify a friend about this article Journal Watch Neurology Summary ... Alert me when this article is cited ISI Web of Science Related Articles Citing Articles (26) Related Articles in Medline Articles in Medline by Author: Glaser, N. Medline Citation Child Health
NEJM -- Risk Factors For Cerebral Edema In Children With Diabetic Ketoacidosis
Risk Factors for cerebral edema in Children with Diabetic Ketoacidosis Nicole Glaser,MD, Peter Barnett, MB, BS, Ian McCaslin, MD, David Nelson, MD, Jennifer
http://content.nejm.org/cgi/content/abstract/344/4/264

Extractions: Nicole Glaser, M.D., Peter Barnett, M.B., B.S., Ian McCaslin, M.D., David Nelson, M.D., Jennifer Trainor, M.D., Jeffrey Louie, M.D., Francine Kaufman, M.D., Kimberly Quayle, M.D., Mark Roback, M.D., Richard Malley, M.D., Nathan Kuppermann, M.D., M.P.H., for The Pediatric Emergency Medicine Collaborative Research Committee of the American Academy of Pediatrics Table of Contents Full Text of this article PDF of this article Editors' Summaries Related editorials in the Journal: Dunger, D. B. Find Similar Articles in the Journal Notify a friend about this article Journal Watch Neurology Summary ... Alert me when this article is cited ISI Web of Science Related Articles Citing Articles (26) Related Articles in Medline Articles in Medline by Author: Glaser, N. Medline Citation Child Health
Annals Of Internal Medicine: Letters
J, Arieff AI. Hyponatremia, cerebral edema, and noncardiogenic pulmonaryedema in marathon runners. Ann Intern Med. 2000;132711
http://www.annals.org/issues/v133n12/full/200012190-00023.html

Extractions: Ayus and colleagues' report of hyponatremic encephalopathy with noncardiogenic pulmonary edema in marathon runners demonstrates the life-threatening potential of this syndrome and its treatment. The authors' use of intravenous hypertonic sodium chloride (514 mmol/L) resulted in an increase in plasma sodium levels (10 mmol/L in 12 hours) to twice the maximal rate recommended as optimal to avoid osmotic demyelination or central pontine myelinolysis (9 mmol/L per 24 hours) . Was there evidence of this lesion at autopsy in the runner with the lowest sodium level (117 mmol/L) who developed brain stem herniation during treatment? Exercise-related hypovolemic hyponatremia, as shown by low pulmonary wedge pressures in this study, might be more appropriately treated with intravenous isotonic sodium chloride (308 mmol/L) to enhance extracellular fluid volume while more gradually correcting plasma tonicity in the high-risk setting of rapid-onset osmotic dysequilibrium . Better understanding of this rare syndrome and potential complications during treatment may lead to safer outcomes and preventive strategies. Arthur J. Siegel, MD
CPS: CPSP Resource Article Cerebral Edema
Recognition and Treatment of cerebral edema Complicating Diabetic KetoacidosisCPSP resource article published September 2000. Investigators
http://www.cps.ca/english/CPSP/Resources/Rcerebraledema.htm

Extractions: Recognition and Treatment of Cerebral Edema Complicating Diabetic Ketoacidosis CPSP resource article published September 2000 Investigators : S. Muirhead, MD, E. Cummings, MD, and D. Daneman, MD Introduction Although early mortality is very low in children and teens with type 1 diabetes, DKA accounts for up to 80% of all deaths. In the largest CE case series (N=69), the outcome was death in 64%, severe disability in 13%, mild disability in 8.6% and intact survival in only 14.5%. Several case reports suggest that subclinical CE may be a common occurrence both before and during DKA treatment. Clinically significant edema may occasionally be present at the time of presentation of the child with DKA to the hospital, but CE generally develops 2 to 24 hours after initiation of DKA treatment. Risk factors for cerebral edema during DKA There is increased representation of and those with new onset diabetes in reports of CE during DKA. There is no convincing evidence that initial acid-base status, glucose, electrolytes or effective osmolality are useful predictors of CE. Thus, all paediatric DKA patients should be presumed to be at risk, independent of initial biochemical parameters.
CPS: Concluded CPSP Studies
cerebral edema in diabetic ketoacidosis. Duration of study. CPSP resource Recognitionand Treatment of cerebral edema Complicating Diabetic Ketoacidosis.
http://www.cps.ca/english/CPSP/Studies/concludedstudies.htm
PetPlace.com - Article: Cerebral Edema
cerebral edema, by Dr. Erika de Papp. Overview cerebral edema is swelling ofthe brain and is most often caused by head trauma in veterinary patients.
http://petplace.netscape.com/articles/artShow.asp?artID=4607
Cerebral Edema
cerebral edema. Increased tissue water in the brain.Return to uscneurosurgery.com Homepage.
http://uscneurosurgery.com/glossary/c/cerebral edema.htm
Radiology Reports Cerebral Edema
Diagnostic reports. NEURORADIOLOGY. cerebral edema. Radiologist's report. figurecerebral edema with obliteration of basal cisterns and lateral ventricle.
http://uscneurosurgery.com/patient education/reports/diagnostic/radiology/crania

Extractions: Neuroradiology Diagnostic reports NEURORADIOLOGY Cerebral edema Radiologist's report figure: cerebral edema with obliteration of basal cisterns and lateral ventricle Description of Ventriculostomy insertion Risks and Complications of Ventriculostomy insertion Clinical Presentation of Cerebral edema Pathophysiology of Cerebral edema ... Homepage
Brain Edema And Cerebral Edema, February 5, 2003
Brain Edema and cerebral edema. cerebral edema is an increase in brain volumecaused by an absolute increase in cerebral tissue water content.
http://www.medical-library.org/journals2a/brain_edema.htm

Extractions: This page has moved. Click here to view. Brain Edema and Cerebral Edema Cerebral edema is an increase in brain volume caused by an absolute increase in cerebral tissue water content. Diffuse cerebral edema may develop soon after head injury. Vasogenic edema arises from transvascular leakage caused by mechanical failure of the tight endothelial junctions of the BBB. Vasogenic edema is frequently associated with focal contusions or hematomas. It eventually resolves as edema fluid is reabsorbed into the vascular space or the brain edema, cerebral edema, adema, brain swelling, herniation Cytotoxic edema is an intracellular process that results from membrane pump failure. It is very common after head injury and is frequently associated with posttraumatic ischemia and tissue hypoxia. Normal membrane pump activity depends on adequate CBF to ensure adequate substrate and oxygen delivery to Consciousness is a state of awareness of the self and of the environment and requires intact functioning of the cerebral cortices and the reticular activating system (RAS) of the brain stem. An altered level of Cerebral herniation occurs when increasing cranial volume and ICP overwhelms the natural compensatory capacities of the CNS. Increased ICP may be the result of posttraumatic brain swelling
HIGH ALTITUDE CEREBRAL EDEMA
HIGH ALTITUDE cerebral edema A serious condition that results fromthe sudden increase in pressure within the brain due to swelling.
http://www.medhelp.org/glossary2/new/GLS_2453.HTM
Cerebral Edema / Swelling Of The Brain Lawsuit News Find Trial
InjuryBoard News cerebral edema / Swelling of the Brain Find trial lawyers andattorneys with experience in lawsuits involving cerebral edema / Swelling of
http://www.injuryboard.com/lvlthreeInjury.cfm/hurl/fstli=5/secli=18/trdli=19
Irish Everest Expedition 2003
High altitude cerebral edema (HACE) is a neurological syndrome resulting from dysfunctionand injury of the central nervous system due to the lack of oxygen at
http://www.irisheverest2003.com/medicine/hace.htm
CEREBRAL EDEMA, INCREASED INTRACRANIAL PRESSURE
cerebral edema, INCREASED INTRACRANIAL PRESSURE Acetazolamide Diamox.Tabs 125 and 250 mg; Sequels, sustained release, 500 mg. For
http://www.neuro.wustl.edu/neuromedical_treatment/edema.html

Extractions: CEREBRAL EDEMA, INCREASED INTRACRANIAL PRESSURE Acetazolamide Diamox. Tabs 125 and 250 mg; Sequels, sustained release, 500 mg. For acute mountain sickness 250 mg q8h, for pseudotumor cerebri 250-750 mg q8h. Dexamethasone Decadron. Tabs 0.25, 0.5, 0.75, 1.5, 4 and 6 mg. Edema associated with neoplasms and encephalitides and in pseudotumor cerebri. Not shown to be of benefit in trauma and infarction edema. 16 mg/24 hrs, divided doses, PO or IV; may be required in doses as high as 100 mg/24hrs. An immediate loading dose of 10 mg IV may be administered in situations considered urgent. When used for several days or longer should be discontinued with gradually smaller doses. Glycerol May be used in the management of acute infarction edema or for many weeks in the management of chronic cerebral edema as in pseudotumor cerebri or brain tumors for which surgery is no longer appropriate. Oral or by nasogastric tube. 1.5-2.5 g/kg/24 hrs, divided doses, 50% solution in 0.9% saline with lemon or orange juice. 1) Meyer,J.S, Churney,J.Z. et al.Treatment with glycerol of cerebral oedema due to acute cerebral infarction. Lancet 2:993-997,1971.
High Altitude Cerebral Edema
First Previous Next Last Index Home Text. Slide 21 of 26.
http://www.samford.edu/schools/education/essm/PHED473/altitudeandillness/sld021.
High Altitude Cerebral Edema
High Altitude cerebral edema (HACE). HACE is swelling of the brain thoughtto be caused by hypoxiadamage to brain tissue. HACE generally
http://www.samford.edu/schools/education/essm/PHED473/altitudeandillness/tsld021
NDI Terminology - Cerebral Edema
cerebral edema. DEFINITION Definition has yet to be entered. Checkback later. The definitions used in this glossary of terminology
http://www.ndif.org/Terms/cerebral_edema.html

Extractions: The definitions used in this glossary of terminology either have been provided by the authors of the articles, or have been extracted wholly or in part, or paraphrased from the following sources: The American Medical Association Encyclopedia of Medicine , Charles B. Clayman, MD, Medical Editor, Random House, New York, 1989; Dorland's Illustrated Medical Dictionary , 28th Edition, W. B. Saunders Company, Philadelphia, 1994; The Random House Dictionary of the English Language , Unabridged Edition, 1966; Webster's Ninth New Collegiate Dictionary

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